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Vagal nerve stimulation has shown promise in attenuating neuroinflammation and improving cognitive outcomes following systemic injuries such as I/R (Wang et al., 2024d)

Likewise, exogenous mitochondrial transplantation restores mitochondrial function, reduces astrocytic and microglial activation, suppresses pro-inflammatory mediators (IL-1, TNF-, COX-2), and enhances hippocampal BDNF expression and neurogenesis, thereby alleviating depressive-like behaviors [106]

Figure 2J] where higher in the BH4 group as compared to the placebo group while free GSH [ F (1, 26) = 7.04, p = 0.01