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Lysosome dysfunction as a cause of neurodegenerative diseases: lessons from frontotemporal dementia and amyotrophic lateral sclerosis

Within this incretin-dominant environment, glucagon receptor activation may contribute more strongly to energy expenditure, fatty acid oxidation, and hepatic lipid metabolism than to uncontrolled hyperglycemia

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In addition to antioxidant and anti-inflammatory properties, HTY has been reported to exhibit antimicrobial properties, potentially through inhibiting the pathogens access to ATP synthase through binding to the ATP synthase polyphenol binding pocket [11] as well as cardioprotective and neuroprotective effects linked to improved endothelial function, lipid oxidation control, and attenuation of oxidative stress [12,13,14,15,16]