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However, despite the widespread use of in vitro and in vivo modelssuch as transgenic animal models and brain organoidsto investigate the effects of natural compounds, these systems often fall short in capturing the full complexity of late-onset Alzheimers disease (LOAD)

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Research indicates BPC-157 significantly enhances cell survival under oxidative stress conditions (H2O2 exposure), protecting fibroblasts during the inflammatory phase of healing when reactive oxygen species are elevated

Activation of oncogenes such as RAS and MYC, along with metabolic reprogramming phenomena like the Warburg effect, further amplify intracellular ROS generation, establishing a reinforcing cycle that perpetuates tumor development [34]