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Several S -glutathiolation mechanisms have been proposed: the direct reaction of GSH with partially oxidized reactive PSHs (thiyl radicals or sulfenic acids), thioldisulfide exchange between PSHs and GSSG or between PSHs and oxidized GSH (sulfenic acid (GSOH)), nucleophilic attack of a protein thiolate on S -nitrosoglutathione (GSNO), and finally, PSHs S -nitrosation followed by S -glutathiolation by GSH to yield the rate of mixed disulfides formation

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To provide more flexibility and reduce burden, we did not propose to require facilities to collect data in a standardized way for the Information Transfer PRO-PM

Targeted pharmacological interventions along these pathways have shown therapeutic promise in models of ischemic brain injury, neurodegeneration, and cancer